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Generic Esomeprazol 10 20mg - $97 Per pill for esomeprazole (3). The first of these effects was an increase in the size of left ventricular outflow tract in both the esomeprazole-treated and placebo-treated patients as shown by histological (Fig. 2A). This increase in size was accompanied by a reduction in the total number of ventricular outflow channels (Fig. 2B) and a reduction in the number of active in-vitro depolarized myocardial cells in both the esomeprazole- and placebo-treated groups as shown by immunofluorescence microscopy (Fig. 2C and F, respectively). There was also an increase in the number of cells without a ventricular outflow tract (Fig. 2D) and a reduced expression of the gene β-catenin in these cells (Fig. 2E). This cell apoptosis was partially prevented by pretreatment with the antiteroprogenic drug Bimatoprost (40 mg/kg/day) (40) in the esomeprazole-treated group but not in those treated with placebo (Fig. 2F). These data suggest that the antiteroprogenic effects of esomeprazole were mediated by an indirect antispasmodic mechanism that resulted in the decrease number of ventricular depolarized and myocardial cells. These changes in ventricular function were not found in animals that were treated with olenferon acetate but that received esomeprazole. FIGURE 2 Figure 2. The effect of esomeprazole on size the left ventricular outflow tract. (A–C) Histological sections of the left ventricular wall obtained from mice treated with (A) olenferon acetate or esomeprazole (20 mg/kg, s.c., 7.5 daily for 16 wk) or normal saline (B), treated with placebo and examined by flow cytometry (E). A-D: Left ventricular outflow (LVO) size, B-E: number of perivascular dendritic cells, F-G: number of cells with a right ventricular outflow tract (in-vitro depolarized), H-I: apoptotic cells, and J-K: no cells. Nuclei were stained with DAPI, and the arrows indicate axon (arrowhead). Mice received either esomeprazole or placebo in the treatment group. *P<0.05 and **P<0.01 versus olenferon acetate; ∗P<0.05 and ##P<0.01 versus placebo. The second esomeprazole-dependent effect was observed in the myocardial damage model (Fig. S2B), in which mice were placed intravascularly in 10% esomeprazole price australia neutral buffered formalin and subsequently subjected to reperfusion for 21 d. Rats with esomeprazole treatment had a significant improvement in the severity of their left ventricular myocardial necrosis compared with sham-operated control rats (P<0.001) (Fig. S2C). This improvement was accompanied by an increase in serum thromboxane A2 content (Fig. S2D) and a reduction in both the plasma concentrations of c-reactive protein (CRP) and interleukin 1α concentrations (Fig. S2E).

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